Uphuhliso lwe-hepatitis kwi-cirrhosis yesibindi, iimpawu.

Okwangoku, oogqirha baninzi bebona izifo zesimo sokugaya abantu, ngokukodwa, isigqeba esinjengobindi. Ngenxa yokusela utywala kunye nokusetyenziswa kweziyobisi, i-hepatitis, izifo ezibangelwa yintlungu, ukuphuhliswa kwesi sifo njenge-cirrhosis yesibindi, iimpawu eziqala ukubonakala ngokukhawuleza.

I-Cirrhosis isifo sesibindi, esiqhubela phambili ngokukhawuleza, ukwakha amaqhinga kunye nokutshintsha izicubu zelungu, kunye nemisebenzi yelungu ngokunyuka kwenyuka, iiseli zayo ziyafa kwaye isibindi sitshabalaliswa. Ngoko i-cirrhosis yesibindi inempawu ezahlukileyo, kwaye zichazwe ngokuxhomekeka kwifom yesifo. Iifom eziqhelekileyo zesifo ziyi-hepatic insufficiency, i-hypertension, umhlaza kunye nezifo ezahlukeneyo ze-bactericidal. Ngelo xesha, i-cirrhosis yesibindi ayimnandi, njengoko ithatha indawo ehamba phambili phakathi kwezifo zenkqubo yokutya, okukhokelela ekufeni. I-cirrhosis ikhula ngokuphindaphindiweyo kumadoda phakathi kweminyaka engamashumi amabini anamanci mathandathu.

Kwimichiza, kuyaqhelekileyo ukuhlukanisa iintlobo ezintathu ze-cirrhosis:

1. I-corn-node. Amanqanaba obuninzi obuhlukeneyo awakhiwe, awanakho ukukhokelela ekuhlaziyweni kwelungu.

2. I-Finite-node. Amacandelo amancinci akhiwa, achaphazela isibindi sonke, kwaye ukuhlaziywa kwakhona kwelungu kunoko kuhamba. Olu hlobo lwesifo luyinto eqhelekileyo yokusela utywala okanye i-anemia.

3. Udibeneyo. Ukuhlaziywa kwesibindi kunye ne-cirrhosis encinci kubangela ukuphuhliswa kwesifo esixubileyo, emva koko ku-cirrhosis enkulu.

Izizathu zokuphuhlisa i-cirrhosis yesibindi, iimpawu zayo, ngokuqinisekileyo, zifana neempawu zesifo se-hepatitis, kubangele izinto ezinjalo: ukungondleki, ukusuleleka, i-granulomatosis, utywala (u-25% one-HIV). Kwaye kunye nezimbangela ze-cirrhosis yi-hepatitis (80% one-HIV) kunye ne-cirphosgenic cirrhosis (i-10% ethelelekileyo), apho kungekho ncazelo malunga nokubonakala kwesifo. Uhlobo olubi kakhulu lwe-cirrhosis luyi-cirrhosis yotywala.

Kwakhona, isifo esinjenge-cirrhosis yesibindi, iimpawu zokubonakala kwazo ziyakubonwa kwiimeko zokuphazamiseka kwentshukumo kunye nokugcinwa kwe-bile, utshintshiselwano obhedu kunye netsimbi, lunethemba elimangalisayo.

Esi sifo sakhiwa ixesha elide, ngelixa izakhi zofuzo zelungu zitshintsha, ukudala iiseli ezintsha ezitshintshileyo zikhula. Kule meko, ukujikeleza kwegazi kunokuphazamiseka kwaye ukuphuma kwegazi kuyancitshiswa, okukhokelela kwimithanjeni ye-varicose yesisu kunye nenye yeengxathu zesisu kunye neengxoworho zangasemva komzimba.

Singaxelela ukuba i-cirrhosis yeempawu zineminye izifo, umzekelo, kunokuba nobuthathaka, ukunyanzelisa, intlungu kwisisu nangamalungu, ukwanda kweqondo lokushisa. Emva koko iimpawu zibonakalisa ngokwabo ngokupheleleyo, njengoko isibindi sishiya ukusebenza. Apha ungabona ukuxilwa, ukutya kunye nokunyanzeliswa kotywala, isisu esicasulayo kunye nokuhlanza, intlungu kwindawo yomzimba ogulayo, ukunyuka okubukhali ekuziphatheni komzimba, i-jaundice kunye nokukhumba isikhumba. Kuzo zonke iimeko, isibindi sanda ngobukhulu.

Ngokuqhelekileyo, iimpawu zesifo zixhomekeke kwisigaba sesifo kunye nomsebenzi wenkqubo yokuvuvukala. Izifundo zibonisa ukuba abantu abangama-60% abantu abanesifo banomfanekiso ocacileyo wokubonakaliswa kwe-cirrhosis, abantu abangama-20% banenkqubela yesi sifo, kwaye i-20% eseleyo ayifumene nesi sifo kwaye siyifumana ngexesha lokuhlolwa kwempilo.

Ngaloo ndlela, iimpawu ze-cirrhosis zesibindi, unyango olunjalo lunceda ukunciphisa ukukhuselwa komzimba olusulelayo, ngoko ke iziyobisi ezichanekileyo kunye nama-immunosuppressants asetyenziswa. Ngonyango olufike ngexesha, umphumo obulalayo awunakwenzeka, nangona kunjalo, ukuba eso sifo siqalisiwe, siphumelela ekuphelelweni kwesibindi kwaye sikhokelela ekufeni kwabantu. Ukwangoku, kukho inani elikhulu leziyobisi ezinokujamelana nesi sifo.